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  1. Inhibition of G6PD by DHEA has been shown to deplete cytosolic glutathione levels, thereby causing contractile dysfunction through dysregulation of Ca2 homeostasis
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  3. Initial, by protein-protein docking, we characterized the protein-protein interface, in which the Cterminal helix of a2C-adrenoceptor is involved in the intricate creation
  4. Initial, the Po of W1282X-CFTR channels in excised FRT patches below control activation conditions was extremely very low similar to our first Po estimates for patches excised from HEK-293T cells
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