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		<title>Angle3oil : Page créée avec « Prompt recognition of severe mycoplasmal infection may allow for earlier treatment and concomitant evaluation of neurologic injury. Pediatr Pulmonol. 2013; 48:98�C101. ?... »</title>
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		<summary type="html">&lt;p&gt;Page créée avec « Prompt recognition of severe mycoplasmal infection may allow for earlier treatment and concomitant evaluation of neurologic injury. Pediatr Pulmonol. 2013; 48:98�C101. ?... »&lt;/p&gt;
&lt;p&gt;&lt;b&gt;Nouvelle page&lt;/b&gt;&lt;/p&gt;&lt;div&gt;Prompt recognition of severe mycoplasmal infection may allow for earlier treatment and concomitant evaluation of neurologic injury. Pediatr Pulmonol. 2013; 48:98�C101. ? 2012 Wiley Periodicals, Inc. &amp;quot;&amp;quot;Flexible bronchoscopy (FB) is the gold standard method of diagnosing tracheomalacia but it is not always feasible in settings with limited resources. Fluoroscopy is sometimes performed as an alternative diagnostic tool but there are no prospective studies that have evaluated the diagnostic accuracy of airway fluoroscopy for tracheomalacia using a-priori definitions. We determined the [http://www.selleckchem.com/products/LY294002.html LY294002] sensitivity, specificity, and likelihood predictive ratio of airway fluoroscopy compared with FB in children suspected of having an airway abnormality. Airway fluoroscopic examination was undertaken within 2-weeks [http://www.selleckchem.com/products/Lapatinib-Ditosylate.html www.selleckchem.com/products/Lapatinib-Ditosylate.html] of a FB in children aged [http://en.wikipedia.org/wiki/Ribosomal_s6_kinase S6 Kinase] cannot replace FB which remains the tool for definitively diagnosing airway malacia. However, in absence of other modalities for diagnosis fluoroscopy should be considered in the setting of persistent respiratory symptoms compatible with the clinical picture of tracheomalacia. Pediatr Pulmonol. 2012; 47:63�C67. ? 2010 Wiley Periodicals, Inc. &amp;quot;&amp;quot;Surfactant deficiency and bronchopulmonary dysplasia (BPD), major obstacles in preterm infants, are addressed with pre- and postnatal glucocorticoids which also evoke harmful catabolic side-effects. Keratinocyte growth factor (KGF) accelerates surfactant production in fetal type II pneumocytes (PN-II), protects epithelia from injury and is deficient in lungs developing BPD, highlighting its potential efficacy in neonates. Neonatal rats were treated with recombinant human (rh)KGF, betamethasone, or their combination for 48?hr prior to sacrifice after which body weight, surfactant, and tissue phosphatidylcholines (PC) were investigated at postnatal d3, d7, d15, and d21.&lt;/div&gt;</summary>
		<author><name>Angle3oil</name></author>	</entry>

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